![]() ![]() This suggests that hippus originates from central PNS activity, and not from SNS activity, or oscillations in the balance between PNS and SNS at the pupil. Pupillary hippus can be extinguished by antagonizing the PNS, whereas agonizing the SNS dilates the pupil without affecting hippus. Finally, pupillary hippus is under-investigated, in particular in children, and our multiple-effectors approach could help shed light on the ANS control of these oscillations. This list may not be comprehensive and may include broader topics as well. ![]() Hippus magnitude (treatment eye relative to control eye) decreased in the TL (-72.8 ± 4.7%, P < 0.0001) and TD (-71.3 ± 2.6%, P < 0.0001) groups, but did not change in the PL (+5.4 ± 13.7%, P = 0.173) group, despite PL pupils dilating to a proportion similar to TD. SNOMED CT: Hippus (75724008) Pupillary athetosis (75724008) These guidelines are articles in PubMed that match specific search criteria developed by MedGen to capture the most relevant practice guidelines. Pupillary hippus with a distinct dominant frequency was present in all measures at baseline (mean: 0.62 Hz, SD: 0.213 Hz), and that frequency did not change in any group (P = 0.971). Hippus, analyzed in both time and frequency domains, was compared between eyes and cohorts. ![]() Measures were taken at baseline, then every 5 minutes for 40 minutes. Bilateral measures of pupil size and dynamics were made over 2.6 seconds using an infrared eye-tracker sampling at 500 Hz. Shifting to a pupil-centered perspective, out of the five studies that discussed pupillary unrest indices or hippus (see Tables 1 and 2 ), only one study reported an effect of. It is concluded that pupillary hippus is unlikely. Specifically, the pupillary outcomes included in this review are: pupil size, pupillary unrest (hippus), pupil size fluctuations, and pupillary light reflex dynamics. Each subject received one drop to the randomly determined treatment eye, while the other eye served as control. The resulting frequency spectrum is very similar to spectra obtained in high luminance non-glaring conditions. We used a paired-eye control study design with three cohorts receiving either 1.0% tropicamide (PNS antagonist) in light (TL), 1.0% tropicamide in dark (TD), or 10% phenylephrine (SNS) in light (PL), n = 12 in each. The purpose of this study was to determine the relative roles of the sympathetic (SNS) and parasympathetic nervous system (PNS) in pupillary hippus. ![]()
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